I scream and cluck with hookteeth and I keep ice on hand for recipients of jeysiec's sick burns but I don't actually hand it out because I'm too busy throwing popcorn at said recipients.
Sometimes NSFW.
Spoilers are tagged: #spoilers
but Supernatural season 9 spoilers are tagged:
#season 9 spoilers
Source: http://wondermumbles.tumblr.com/post/57965880214
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Case in point is Facebook's more recent push to get more celebrities to use the site more publicly. That's an obvious response to Twitter's success in the area and, while Facebook may never be able to directly compete with the former, it's trying to attract at least some of the activity normally reserved for Twitter.
All Things D is underlining this fact with some inside knowledge from Facebook, though the social network won't say publicly that it's interested in driving people away from Twitter.
But there's good reason to be interested. Facebook's ad revenue keeps on growing and it's already the second largest player in some areas, behind Google.
However, Twitter is getting a lot of attention from the entertainment business, TV, movies, and music. And that attention translates into cold hard cash.
Source: http://news.softpedia.com/news/Facebook-Wants-Steal-Celebrities-Away-from-Twitter-374020.shtml
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NCI Backgrounder
Being the third most common non-skin cancer and the third leading cause of cancer-related mortality in men and women in the United States, colorectal cancer can be a devastating disease. Nonetheless, according to the latest statistics from NCI covering the period 2006 to 2010, death rates continue to decline, due in part to improved treatments, and in part to colorectal cancer screening options that are proven to reduce mortality and that have gained acceptance by the public.
Colorectal cancer can still be a deadly disease, and people who have been diagnosed and treated for it are at increased risk for developing a new colon cancer. To address concerns of colorectal cancer recurring after initial treatment, NCI, in collaboration with SWOG, one of the five cooperative groups that together will comprise NCI's National Clinical Trials Network, and Cancer Prevention Pharmaceuticals, Inc., recently announced a phase III trial. The Preventing Adenomas of the Colon with Eflornithine & Sulinidac (PACES) trial looks at whether someone who has been treated for colon cancer in the past can lower his or her risk of having a second primary colorectal cancer or an adenoma (a type of polyp on the colon wall that may be precancerous) by regularly taking one or both of the study drugs, eflornithine and sulindac. ?Both drugs work to lower the body's levels of polyamines, a group of naturally-formed molecules that play a role in the development of colorectal cancer. Each drug works in a different manner: eflornithine slows the body's production of polyamines; sulindac helps cells get rid of excess polyamines.
?PACES is an important study that, in addition to studying ways to prevent the recurrence of previously treated colon cancer, will use new genomic technology to help identify those patients with the greatest risk of recurrence,? said Worta McCaskill-Stevens, M.D., chief of the Community Oncology and Prevention Trials Research Group in NCI?s Division of Cancer Prevention.
Researchers chose these two drugs because of an earlier study that looked at their preventive effects in patients who already had at least one adenoma removed from their colon. In that study, participants who took the drug combination lowered their risk of developing another adenoma over the next three years to less than one third of what it was for those who did not take the drugs. And they lowered their chances of developing high-risk adenomas or multiple adenomas during that time by 90 percent. According to McCaskill-Stevens, ?this trial will help determine who will most likely benefit from the drugs eflornithine and sulindac.?
SWOG (formerly the Southwest Oncology Group) conducts clinical trials to prevent and treat cancer in adults and is primarily funded by research grants from NCI. It is one of the largest cancer clinical trials cooperative groups in the United States.
Source: http://feedproxy.google.com/~r/ncinewsreleases/~3/dCumsb4lOYA/PACEStrial
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The Veery thrush, a secretive migratory bird, silences its flute-like twilight song when owls are around to avoid getting eaten, a new study shows.
The trembling, other-worldly dusk chorus of the Veery thrush?(Catharus fuscescens) is often heard well after sunset. But all this singing in low light can be dangerous for the small brown and white birds. Perching makes them less vigilant and their racket can give away their exact location to predators like owls that just beginning their nocturnal hunt.
But Veeries adjust their behavior to be less conspicuous, the new study found.
Researchers studied a group of the birds in a stretch of forest in Millbrook, N.Y., a home shared with up to three pairs of predatory barred owls. When the scientists played recordings of the owl songs in the forest, Veeries cut their singing patterns for up to 30 minutes. The birds also had fewer song bouts at dusk and quit singing earlier in the night after having heard the owls.?
"Singing becomes much more risky in the low light of dusk when owls are around," study researcher Kenneth Schmidt of Texas Tech University said in a statement. "However, by eavesdropping on owls, Veeries can adapt their singing behavior to decrease the risk of predation."
"Further studies of dusk chorus singing may reveal how the risk of being attacked by predators has contributed to the evolution of singing behavior at dusk," Schmidt added.
The research is detailed in journal Behavioral Ecology and Sociobiology.
Follow Megan Gannon on Twitter and Google+. Follow us @livescience, Facebook & Google+. Original article on LiveScience.com.
Copyright 2013 LiveScience, a TechMediaNetwork company. All rights reserved. This material may not be published, broadcast, rewritten or redistributed.Source: http://news.yahoo.com/owl-hoots-hush-song-thrush-135211494.html
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Source: http://www.facebook.com/novumadventum/posts/10151615393007865
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